Why Does Your Nose Get Stuffy One Nostril at a Time?

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Because your nostrils split their workload. Throughout the day, they each take breaks in a process of alternating congestion and decongestion called the nasal cycle. At a given moment, if you're breathing through your nose, the lion’s share of the air is going in and out of one nostril, with a much smaller amount passing through the other. Every few hours, your autonomic nervous system, which takes care of your heart rate, digestion and other things you don’t consciously control, switches things up and your other nostril does all the heavy lifting for a little while.  The opening and closing of the two passages is done by swelling and deflating erectile tissue - the same stuff that’s at work when your reproductive organs are aroused - up in your nose.

The nasal cycle is going on all the time, but when you’re sick and really congested, the extra mucous often makes the nostril that’s on break feel much more backed up.

There are at least two good reasons why nasal cycling happens.

One, it makes our sense of smell more complete. Different scent molecules degrade at different rates, and our scent receptors pick up on them accordingly. Some smells are easier to detect and process in a fast-moving airstream like the decongested nostril, while others are better detected in the slower airstream of the congested nostril. Nasal cycling also seems to keep the nose maintained for its function as an air filter and humidifier. The alternating congestion gives the mucous and cilia (the tiny hairs up in your nose) in each nostril a well-deserved break from the onslaught of air and prevents the insides of your nostrils from drying out, cracking and bleeding.

Another snot mystery: Why does my nose run when I cry?

When you cry, most of your tears spill over your lower eyelid and run down your face. Some of them, though, don’t quite make it over the hill and instead go back down into the tear ducts and into the nasal cavity, which is connected to the ducts. If you’re really bawling, you’ll have quite a few tears running down into the nose, and their salinity also helps loosen up mucous and get it flowing, giving you a runny nose.

Pandemic vs. Epidemic: What’s the Difference?

If scientists can't develop a vaccine for a new virus quickly enough, an epidemic can turn into a pandemic.
If scientists can't develop a vaccine for a new virus quickly enough, an epidemic can turn into a pandemic.
doble-d/iStock via Getty Images

As the new coronavirus continues to spread around the world, the words epidemic and pandemic are showing up in news reports more often than they usually do. While the terms are closely related, they don’t refer to the same thing.

As the Association for Professionals in Infection Control and Epidemiology (APIC) explains on its website, “an epidemic occurs when an infectious disease spreads rapidly to many people.” Usually, what precedes an epidemic is an outbreak, or “a sudden rise in the number of cases of a disease.” An outbreak can affect a single community or several countries, but it’s on a much smaller scale than an epidemic.

If an epidemic can’t be contained and keeps expanding its reach, public health officials might start calling it a pandemic, which means it’s affected enough people in different areas of the world to be considered a global outbreak. In short, a pandemic is a worldwide epidemic. It infects more people, causes more deaths, and can also have widespread social and economic repercussions. The spread of the Spanish influenza from 1918 to 1919, which killed between 20 and 40 million people around the world, was a pandemic; more recently, the H1N1 influenza created a pandemic in 2009.

Here’s where it gets a little tricky: There’s no cut-and-dried classification system for outbreaks, epidemics, and pandemics. Based on the definitions above, it might seem like the current coronavirus disease, now called COVID-19, falls into the pandemic category already—according to a map from the World Health Organization (WHO), there are more than 80,000 confirmed cases in 34 countries, and nearly 2700 people have died from the disease. It’s also beginning to impact travel, stock markets, and the global economy as a whole. But WHO maintains that although the situation has the potential to become a pandemic, it’s still an epidemic for now.

“It really is borderline semantics, to be honest with you,” Anthony Fauci, director of the National Institute of Allergy and Infectious Diseases, told CNN earlier this month. “I think you could have people arguing each end of it. Pandemics mean different things to different people.”

[h/t APIC.org]

Fat Bats Might Be Resistant to Deadly White-Nose Syndrome

Penn State, Flickr // CC BY-NC-ND 2.0
Penn State, Flickr // CC BY-NC-ND 2.0

Good news for flying mammals: chubby little brown bats might be genetically resistant to white-nose syndrome, a fungal disease that’s killed more than 5.5 million bats since it was first documented in 2006 [PDF]. A new study in the journal Scientific Reports describes three genetic adaptations in the bats that could protect them from the pathogen.

Little brown bats (Myotis lucifugus), common in Canada and the eastern United States, are especially susceptible to white-nose syndrome. According to lead author Giorgia G. Auteri, a doctoral candidate at the University of Michigan, white-nose syndrome kills bats by disrupting their hibernation cycles.

“When they’re in hibernation in the winter, they’re not meant to be waking up. They’re supposed to be asleep,” Auteri tells Mental Floss. “But this fungus grows on them, and it causes the bats to keep waking up during hibernation. And because they’re waking up when they shouldn’t be, they’re running out of fat reserves too early.”

But while white-nose syndrome has devastated bat populations in North America, not all infected bats die from the disease—some recover. Auteri wanted to find out what made the survivors so special.

Auteri and her team compared the genetic makeup of nine surviving and 29 non-surviving little brown bats from northern Michigan. They discovered that survivors share three important genetic distinctions. “One is involved with fat metabolism,” she says. “And another is involved with regulating when the bats wake up from hibernation. And the third gene is involved in their echolocation ability, in their sonar for hunting insects.”

The results make sense, Auteri says. Because white-nose syndrome interrupts bats’ hibernation schedules, bats with genes that relate to more optimal fat storage (i.e., they’re fatter) and better hibernation regulation (i.e., they sleep longer) are more likely to survive the disease.

Auteri’s research could help scientists and conservationists find ways to preserve little brown bat populations. Besides being adorable, little brown bats also play an important ecological role as predators of insects like mosquitoes, moths, and other pests that are destructive to crops and forests.

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